|Posterior drawer test for knee instability
The question I have asked myself over the years is: Why do these joints become unstable?
About 20 years ago, a patient came to see me regarding a condition of low back pain. He also happened to mention that he was scheduled for knee surgery to repair the ACL. As part of my examination, I checked his knee stability. I noticed that it was extremely unstable, but thought nothing more of it. I proceeded to treat him using a new technique I had been developing, called Matrix Repatterning (MR). I found a number of injury patterns associated with the lower extremities, the pelvis and the thoracolumbar spine and rib cage, and treated these accordingly. A few weeks later, after a series of about six sessions, the patient reported that his back pain was gone. He also happened to mention that his knee also felt much better.
“As a matter of fact,” he stated, “when I went back for my pre-operative examination with the surgeon, he checked my knee and couldn’t find anything wrong with it anymore. He said that he didn’t know why I was told I needed surgery in the first place.”
I didn’t know how to respond, but decided to go ahead and recheck his knee stability. It was perfect.
That case got me thinking. How had his knee regained stability? Did my treatment have anything to do with the improvement? I started monitoring knee stability in my patients, and noticed that it often improved when I treated certain areas of the body. Slowly, a pattern began to emerge demonstrating a rather consistent association with injuries in other areas of the body that somehow seemed to be associated with knee instability. Once these areas of focal tissue injury – which I refer to as primary restrictions – were resolved, unstable knees would become stable – and stay that way. At the time, other than observing this rather startling clinical manifestation, I had no way to explain the mechanism of improvement.
New theory of joint stability
Several years later, a podiatrist came to see me for his chronic knee pain. As I performed my initial assessment, I was able to demonstrate how his knee instability was directly associated with an injury to his pelvis. As I positioned the electronic device (MatrixPulse Scanner) used to determine the location of primary restrictions on his pelvis, he was quite astonished that I was able to immediately restore the knee stability.
“How was that possible?” he asked. In the past, I had considered some type of compensatory alteration in the tensile properties of the ACL and PCL. But this was an immediate change, and I could not understand how ligaments that had been lax could suddenly increase in tone. And, here was a fellow health professional, someone highly respected in the field of podiatric medicine, and who worked closely with a number of orthopedic surgeons; I needed to consider my explanation carefully.
I considered the evidence:
- The knee was initially profoundly unstable.
- I temporarily neutralized the primary restriction in his pelvis, using the scanner.
- The knee instantly stabilized.
- I removed the scanner.
- The knee once again became unstable – instantly.
Recent studies have postulated the popliteus as an important dynamic stabilizer of the knee. It is intricately attached to several internal structures of the knee, including the lateral meniscus, the posterior capsule of the knee, the PCL and the MCL. I decided to look into the possible role of this muscle on the function of the knee. I also investigated the possible role of appropriately targeted treatment, such as Matrix Repatterning, which might help restore stability to the knee, and thus promote healing of this joint.
The typical clinical approach to an unstable knee is through exercise of the large muscle groups, or, if all else fails, surgery for the so-called “torn ACL or PCL.” The purported incidence of actual tears of these ligaments appears to be much less than indicated by radiological reports. Surgical reports often contradict the findings of radiologists in these cases. Recently, I have also discovered that anterior instability of the knee is often accompanied by reduced tone of the biceps femoris, and that treatment of the pelvis often restores tone in this muscle along with knee stability, as measured with an anterior drawer test. Additional muscular and fascial associations appear to mediate ankle stability. In the shoulder, a similar mechanism has been identified regarding the possible role of the supraspinatus as a stabilizer of the gleno-humeral joint.
Symptom of core injury
The Matrix Repatterning assessment is designed to determine the location of focal areas of tissue restriction, referred to as primary restrictions (PRs). PRs are often located in areas of the body remote from the area of symptoms, which may be compensatory to the effects of the PRs. As I reviewed the areas containing the primary restrictions that seemed to be associated with joint instability, I noticed that they were invariably located in the torso and pelvis – the so-called core structures of the body.
Treatment of these areas usually resulted in restoration of stability in the involved joints, which appeared to occur primarily in peripheral joints. The one exception was the lumbar spine, which is located in the core region. But this one exception proved to be a key to my understanding of this important mechanism.
|Figure 1: sacrificial joints (red) and core structures (blue)
Could the unstable joints, in fact, be sacrificial joints (see Figure 1)? What about the lower lumbar spine? The spinal cord is a key core structure that must be protected. However, as we know, the spinal cord ends at the level of L2 or L3. Thus, the neurological structures of the lower lumbar spine (cauda equina) – composed of separate nerve tracts, each protected by the meninges – are much less vulnerable to serious damage. People with lower lumbar injury tend to have much less debilitating impairment than those with direct spinal cord injuries. Thus, the response of the lower lumbar spine fits the model of instability that I had been formulating.
Turning joint stability back on
Matrix Repatterning is a gentle form of manual therapy, based on a recent understanding of the ultrastructure of the body, specifically the intracellular and extracellular matrix, and their influence on structural, mechanical and physiological properties of the body. The basic premise is that gentle manual therapy applied to areas of the body identified as primary restrictions can help to restore the functional capacity of tissue, reinstating its elastic and electrochemical properties. Primary restrictions are often located in the deep core structures within the skeletal frame (referred to as intraosseous lesions), and the fluid-filled thoracic and abdomino-pelvic areas, directly related to the spine, pelvis and rib cage. These injury patterns are often associated with impact injuries, such as falls, sports injuries and motor vehicle collisions.
Remarkably, treatment of primary restrictions, often located in remote parts of the body, using Matrix Repatterning, appears to immediately restore stability in certain joints. What could account for this response? It is my theory that there may be a feedback mechanism, mediated by spinal reflexes or mechanical-electro-chemical signals generated within the extracellular matrix (ECM). These systems may be able to monitor potentially threatening injuries, and respond by triggering inhibitory or excitatory efferent neurons (spinal/neurological level), or via the production of electro-chemical signals to activate specific tonal structural proteins within the ECM.
Whatever the mechanism may be, it appears to allow the body to rapidly modulate stability of the peripheral, appendicular joints, as well as – notably – the lower lumbar spine.
Practitioners, trained in Matrix Repatterning, have confirmed that joint stability is often restored when the underlying primary restrictions are properly addressed. In this way, they have been able to help many conditions associated with joint instability, resulting in significant clinical improvement.
Further research is also required to assess the effectiveness of this approach on the possible reversal or prevention of degenerative changes associated with these joints.